In a child who has ingested a beta blocker, which metabolic disturbance is most likely?

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Multiple Choice

In a child who has ingested a beta blocker, which metabolic disturbance is most likely?

Explanation:
Beta-adrenergic signaling is a key driver of hepatic glucose production during fasting, promoting glycogenolysis and gluconeogenesis. When a child swallows a beta blocker, this adrenergic drive is blocked, so the liver can’t release glucose as effectively. In a fasting or ill child, this setup commonly leads to acute hypoglycemia. Hyperglycemia, hyperkalemia, and hyponatremia aren’t typical primary effects of beta-blocker overdose in children, though the blockade can mask early hypoglycemia signs, making glucose testing especially important.

Beta-adrenergic signaling is a key driver of hepatic glucose production during fasting, promoting glycogenolysis and gluconeogenesis. When a child swallows a beta blocker, this adrenergic drive is blocked, so the liver can’t release glucose as effectively. In a fasting or ill child, this setup commonly leads to acute hypoglycemia. Hyperglycemia, hyperkalemia, and hyponatremia aren’t typical primary effects of beta-blocker overdose in children, though the blockade can mask early hypoglycemia signs, making glucose testing especially important.

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